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Excessive dietary fructose consumption appears to contribute to metabolic syndrome and non-alcholic fatty liver disease.

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←Previous: excessive dietary fructose consumption may underlie the development of nonalcoholic fatty liver disease and the metabolic syndrome.

Next: Fructose supplementation induced hypertriglyceridemia and hyperleptinemia, hepatic steatosis and necroinflammation, increased the expression of genes related to fatty acid synthesis and decreased fatty acidβ-oxidation activity.→

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